TUDCA (Tauroursodeoxycholic Acid) vs Urolithin A

Side-by-side comparison of mechanisms, dosing, interactions, and stacking potential.

TUDCA (Tauroursodeoxycholic Acid)Urolithin A
CategorySupplementsSupplements
Standard Dose250-500mg daily
TimingWith meals for liver support. Some protocols recommend before meals to prime bile flow.
Cycle DurationCycle 4-8 weeks for liver support; ongoing at low dose for chronic liver conditions under supervision
Evidence LevelModerate-StrongModerate-Strong

Mechanism

TUDCA is a water-soluble bile acid conjugate (taurine + ursodeoxycholic acid) with potent hepatoprotective and cytoprotective properties. It inhibits the mitochondrial pathway of apoptosis by preventing BAX translocation to mitochondria and cytochrome c release. TUDCA reduces endoplasmic reticulum (ER) stress by acting as a chemical chaperone, assisting protein folding and reducing the unfolded protein response (UPR). It protects hepatocytes by stabilizing cell membranes, displacing toxic hydrophobic bile acids (chenodeoxycholic, lithocholic) from biliary epithelium, and promoting choleresis (bile flow). Additionally, it has neuroprotective effects via reduction of ER stress in neurons and modulation of the TGR5 bile acid receptor.

Standard Dosing

250-500mg daily

Timing

With meals for liver support. Some protocols recommend before meals to prime bile flow.

Cycle Duration

Cycle 4-8 weeks for liver support; ongoing at low dose for chronic liver conditions under supervision

Side Effects

  • Diarrhea (dose-dependent)
  • GI discomfort
  • Flatulence
  • Rare: calcification of gallstones

Contraindications

  • Complete biliary obstruction (cannot reach bile ducts if obstruction is complete)
  • Calcified gallstones (UDCA/TUDCA dissolve cholesterol stones, not calcified)
  • Acute cholangitis
  • Complete biliary obstruction
  • Gallstones (may mobilize stones)

Best Stacking Partners

Milk Thistle (Silymarin)NACAlpha Lipoic AcidArtichoke Extract
B

Urolithin A

Supplements

Mechanism

Gut metabolite of ellagitannins that activates mitophagy — selective clearance of damaged mitochondria. Improves mitochondrial function, muscle endurance, and cellular energy production. Only ~40% of population can produce it endogenously.

Contraindications

  • Limited long-term safety data beyond 4 months

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